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窦房

网络sinus

窦房

窦房

sinus

...掌握的，耳朵前侧的先天性廔管 (fistula)、窦房 (sinus)、凹陷 (pit) 等情形，可能是因为胚胎发育时，组织构造的融合不完整 …

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Your SA node sends out the electrical signal that guides the rest of the heart tissue .

你的窦房结发出的电信号会指导心脏的其他组织。

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is caused by the nonlinear interaction between the sinus node and myopic Ventricular focus in the heart .

心脏窦房结与异位心室搏动非线性相互作用，导致调制并行收缩反常节律。

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Activation of the heart normally begins in the sinoatrial node .

在正常情况下，心脏的活动始于窦房结。

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These cell clusters in the central part of the sinus node are all around the sinus node artery .

窦房结中心区的细胞集落，全都存在于窦房结动脉周围。

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Objective To understand the clinical value of Holter ECG monitoring diagnose SSS.

目的为了明确动态心电图对病态窦房结综合征的诊断价值。

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The signals that make the heart 's muscle fibres contract come from the sinoatrial node , which is the natural pacemaker of the heart .

驱使心脏肌肉纤维收缩的信号来自于窦房结，它是心脏天然的起搏器。

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An increase in extracellular potassium does in fact shift the pacemaker within the sinus node from the cephalic to the caudal portion.

细胞外液钾的增高确实能使窦房结内的起博点从头侧移向尾侧。

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Research on the main syndrome manifestation of SSS before and after it's cured by heart pacemaker .

研究病态窦房结综合征患者在心脏起搏治疗后的主要中医证候；

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The sinoatrial node is located at the junction of the superior vena cava and the right atrium .

窦房结位于上腔静脉与右心房的交界处。

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This process continues over and over as the atria refill with blood and other electrical signals come from the SA node .

随着心房的再次充血和窦房结电信号的再次产生，这个过程会一直重复下去。

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Stressor load is modeled by fast neuronal imputation arriving on sinoatrial node .

压力负荷模型的快速神经对窦房结抵达估算。

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The sluggish of antrum room block of provisionality sees at vagus tension heighten .

暂时性的窦房阻滞见于迷走神经张力增高。

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Recent studies show that human SAN pace-making gene HCN4 mutations are closely associated with sick sinus syndrome .

近年来有较多研究表明，人窦房结起搏基因 HCN4 突变与病态窦房结综合征密切相关。

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Clinical Study on Treating Sick Sinus Syndrome With Tiaolu Solution ?

调律冲剂治疗病态窦房结综合征的临床研究？。

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Results : The SAN and AVN chiefly consisted of P cells and T cells .

结果：窦房结和房室结内的细胞主要为 P 细胞和 T 细胞。

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This is particularly important when the conducting tissues between the SA node and the ventricles fail only from time to time .

尤其在窦房结和心房之间的传导组织有时发生故障时这点尤为重要。

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It briefly describes the nature of excitable media, the knowledge of Sinus and cardiac pacemakers, as well as cellular automata and so on .

简要介绍了激发介质的性质、窦房结和心脏起搏器的有关知识、以及元胞自动机等内容。

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When the sinoatrial node starts to malfunction it can cause dangerously slow or erratic rhythms to develop .

窦房结出了问题，心律会变缓或变紊乱，这很危险。

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